Deficient aversive-potentiated startle and the triarchic model of psychopathy: The role of boldness
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Título
Deficient aversive-potentiated startle and the triarchic model of psychopathy: The role of boldnessFecha de publicación
2016-05xmlui.dri2xhtml.METS-1.0.item-edition
PostprintEditor
ElsevierCita bibliográfica
ESTELLER, Àngels; POY GIL, Roser; MOLTÓ BROTONS, Javier. Deficient aversive-potentiated startle and the triarchic model of psychopathy: The role of boldness. Biological Psychology (2016), v. 117, pp. 131-140Tipo de documento
info:eu-repo/semantics/articleVersión de la editorial
http://www.sciencedirect.com/science/article/pii/S0301051116300904Versión
info:eu-repo/semantics/acceptedVersionPalabras clave / Materias
Resumen
This study examined the contribution of the phenotypic domains of boldness, meanness, and disinhibition of the triarchic conceptualization of psychopathy (Patrick, Fowles, & Krueger, 2009) to deficient aversive-pote ... [+]
This study examined the contribution of the phenotypic domains of boldness, meanness, and disinhibition of the triarchic conceptualization of psychopathy (Patrick, Fowles, & Krueger, 2009) to deficient aversive-potentiated startle in a mixed-gender sample of 180 undergraduates. Eyeblink responses to noise probes were recorded during a passive picture-viewing task (erotica, neutral, threat, and mutilation). Deficient threat vs. neutral potentiation was uniquely related to increased boldness scores, thus suggesting that the diminished defensive reaction to aversive stimulation is specifically linked to the charm, social potency and venturesomeness features of psychopathy (boldness), but not to features such as callousness, coldheartedness and cruelty traits (meanness), even though both phenotypes theoretically share the same underlying low-fear disposition. Our findings provide further evidence of the differential association between distinct psychopathy components and deficits in defensive reactivity and strongly support the validity of the triarchic model of psychopathy in disentangling the etiology of this personality disorder. [-]
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Biological Psychology (2016), v. 117Derechos de acceso
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info:eu-repo/semantics/openAccess
info:eu-repo/semantics/openAccess
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